Jane Stewart (scientist)

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Jane Stewart
Alma materQueen's University,
University of London
Scientific career
FieldsBehavioral neuroscience
InstitutionsConcordia University

Jane Stewart OC FRSC is a Canadian neuroscientist who has been active in the fields of psychology, psychiatry, and psychopharmacology. She is a professor emerita at Concordia University in Montreal, Canada.

Career[]

Stewart earned a Bachelor of Arts degree in psychology and biology from Queen's University in 1956, and PhD in psychology in 1959 from the University of London, England.[1][2] She then started working for Ayerst Pharmaceuticals in Montreal and subsequently joined Concordia University in 1962,[3] where she served as chair of the Department of Psychology (1969–1974) and director of the Center for Studies in Behavioral Neurobiology (1990–1997).[1] She served on many grant review committees and on the editorial boards of 11 peer-reviewed scientific journals.[1]

Research[]

Stewart has made seminal contributions to different areas of research, such as conditioned drug effects,[4][5] the motivational effects of drugs,[6] circadian rhythms,[7] antidepressant and antipsychotic drug action,[8][9] and sexual behavior.[10][11]

Honors[]

Stewart was awarded an honorary degree from Queen's University and is a Fellow of the American Association for the Advancement of Science, the American Psychological Association, the Canadian Psychological Association, and the Royal Society of Canada.[1] She also received the highest civilian honor in her country, being appointed Officer in the Order of Canada in 2007.[1] A special issue of the journal Biological Psychiatry was dedicated to her on the occasion of her retirement in 2008.[1]

Significant papers[]

  • Kalivas PW, Stewart J (1991). "Dopamine transmission in the initiation and expression of drug- and stress-induced sensitization of motor activity". Brain Research Reviews. 16 (3): 223–44. doi:10.1016/0165-0173(91)90007-U. PMID 1665095. (cited over 1300 times)[12]
  • Stewart J, de Wit H, Eikelboom R (April 1984). "Role of unconditioned and conditioned drug effects in the self-administration of opiates and stimulants". Psychological Review. 91 (2): 251–68. doi:10.1037/0033-295X.91.2.251. PMID 6571424. (cited over 600 times)[12]
  • de Wit H, Stewart J (1981). "Reinstatement of cocaine-reinforced responding in the rat" (PDF). Psychopharmacology. 75 (2): 134–43. doi:10.1007/BF00432175. PMID 6798603. (cited over 400 times)[12]
  • Stewart J, Badiani A (1993). "Tolerance and sensitization to the behavioral effects of drugs". Behavioural Pharmacology. 4 (4): 289–312. doi:10.1097/00008877-199308000-00003. PMID 11224198. (cited over 350 times)[12]
  • Shaham Y, Shalev U, Lu L, De Wit H, Stewart J (July 2003). "The reinstatement model of drug relapse: history, methodology and major findings". Psychopharmacology. 168 (1–2): 3–20. doi:10.1007/s00213-002-1224-x. PMID 12402102. (cited over 300 times)[12]

References[]

  1. ^ a b c d e f de Wit H, Shaham Y (May 2009). "Incentive motivation, conditioning, stress, and neuropsychiatric disorders: A tribute to Jane Stewart". Biological Psychiatry. 65 (10): 827–8. doi:10.1016/j.biopsych.2008.12.012. PMC 2716031. PMID 19398047.
  2. ^ "Jane Stewart CV" (PDF). Concordia University. 2015. Retrieved 2019-12-13.
  3. ^ "Jane Stewart". www.concordia.ca. Retrieved 2016-03-09.
  4. ^ Eikelboom R, Stewart J (September 1982). "Conditioning of drug-induced physiological responses". Psychological Review. 89 (5): 507–28. doi:10.1037/0033-295X.89.5.507. PMID 7178331.
  5. ^ Stewart J (June 1992). "Neurobiology of conditioning to drugs of abuse". Annals of the New York Academy of Sciences. 654: 335–46. doi:10.1111/j.1749-6632.1992.tb25979.x. PMID 1321575.
  6. ^ Stewart J, de Wit H, Eikelboom R (April 1984). "Role of unconditioned and conditioned drug effects in the self-administration of opiates and stimulants". Psychological Review. 91 (2): 251–68. doi:10.1037/0033-295X.91.2.251. PMID 6571424.
  7. ^ Amir S, Stewart J (February 1996). "Resetting of the circadian clock by a conditioned stimulus". Nature. 379 (6565): 542–5. doi:10.1038/379542a0. PMID 8596633.
  8. ^ Stewart J, Rajabi H (August 1996). "Initial increases in extracellular dopamine in the ventral tegmental area provide a mechanism for the development of desipramine-induced sensitization within the midbrain dopamine system". Synapse. 23 (4): 258–64. doi:10.1002/(SICI)1098-2396(199608)23:4<258::AID-SYN3>3.0.CO;2-6. PMID 8855510.
  9. ^ Samaha AN, Seeman P, Stewart J, Rajabi H, Kapur S (March 2007). ""Breakthrough" dopamine supersensitivity during ongoing antipsychotic treatment leads to treatment failure over time". Journal of Neuroscience. 27 (11): 2979–86. doi:10.1523/JNEUROSCI.5416-06.2007. PMC 6672560. PMID 17360921.
  10. ^ Mitchell JB, Stewart J (March 1990). "Facilitation of sexual behaviors in the male rat associated with intra-VTA injections of opiates". Pharmacology Biochemistry and Behavior. 35 (3): 643–50. doi:10.1016/0091-3057(90)90302-X. PMID 1971113.
  11. ^ Mitchell JB, Stewart J (February 1990). "Facilitation of sexual behaviors in the male rat in the presence of stimuli previously paired with systemic injections of morphine". Pharmacology Biochemistry and Behavior. 35 (2): 367–72. doi:10.1016/0091-3057(90)90171-D. PMID 2320644.
  12. ^ a b c d e Web of Science, accessed May 6, 2009
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